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Product Name: CDK5 Antibody [1H3]
Species Reactivity: Human, Mouse, Rat
Tested Applications: IHC, WB
Applications:
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight: 28
Immunogen: Purified rat Cdk5
Host Species: Mouse
Purification: Protein G purified
Physical State: Liquid
CAS NO.: 24169-02-6
Product: Econazole (nitrate)
Buffer: 100 uL in 10 mM HEPES (pH 7.5), 150 mM NaCl, 100 ug per mL BSA and 50% glycerol.
Concentration:
Storage Conditions:
Clonality: Monoclonal
Conjugate: Unconjugated
Alternate Names: Cdkn5, Cell division protein kinase 5,
Accession NO.: Q03114
Protein Ino: 416783
Official Symbol: Cdk5
Geneid: 140908
Background:
PubMed ID:http://aac.asm.org/content/35/8/1679.abstract

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Title Loaded From File
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Aranodes, and juxtaparanodes. Alterations ofthe axo-glial interaction contribute to the etiology of many neurological diseases.
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The mechanisms underlying the lower in severity of CIA following administration of GMSCs. GMSC injection
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Es Sp1-6 and Sp1-7) was deleted, an further reduction in luciferase activity was observed. These
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Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Sembly, 2014 (un.org/en/ga/search/view_doc.asp?symbol=A/ RES/69/1, accessed 25 November 2014). 5. Statistical Tables on Overseas Filipino Workers
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Of S. spinosa Lu106 exhibited a development defect relative to that with the wild form.
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Assay from the studied drugs in pure forms and pharmaceutical formulations.Conflicts of InterestsThere is no
Autophagy. Therefore we conclude that vacuolar lipase activity is, for by far the most element,
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Nto the pathogenesis of aortic stenosis.AcknowledgmentsFunded by grants from the American Heart Association (AHA: 11GRNT7900016)
G of oxidative phosphorylation, high amplitude mitochondrial swelling and outer membrane rupture (Di Lisa et
That A-ring modifications seem to become tolerable for yielding biologically intriguingThat A-ring modifications seem to
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Ther up-regulated in prostate cancer [9], also as non-prostatic malignancies including gastric cancer [10]. PSCA
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Ysis of pheromone-dependent gene transcription in WT and reg1 cells. CellsYsis of pheromone-dependent gene transcription
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Sets we discover that there is a statistical difference (P = 2.eight ?1026), confirming that
Te-sex pheromonal odors: 6-OHDA lesions of DA terminals within this area abolished the hardwired D1
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