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Product Name: HEPACAM2 Antibody
Species Reactivity: Human, Mouse, Rat
Tested Applications: ELISA, WB
Applications: HEPACAM2 antibody can be used for detection of HEPACAM2 by Western blot at 0.5 – 1 μg/mL.
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight: 51 kDa
Immunogen: Rabbit polyclonal HEPACAM2 antibody was raised against a 19 amino acid peptide near the carboxy terminus of human HEPACAM2.The immunogen is located within amino acids 370 – 420 of HEPACAM2.
Host Species: Rabbit
Purification: HEPACAM2 Antibody is affinity chromatography purified via peptide column.
Physical State: Liquid
CAS NO.: 487-39-8
Product: GSK682753A
Buffer: HEPACAM2 Antibody is supplied in PBS containing 0.02% sodium azide.
Concentration: 1 mg/mL
Storage Conditions: HEPACAM2 antibody can be stored at 4˚C for three months and -20˚C, stable for up to one year. As with all antibodies care should be taken to avoid repeated freeze thaw cycles. Antibodies should not be exposed to prolonged high temperatures.
Clonality: Polyclonal
Conjugate: Unconjugated
Alternate Names: HEPACAM2 Antibody: MIKI, MIKI, UNQ305/PRO346, HEPACAM family member 2, Mitotic kinetics regulator
Accession NO.: NP_001034461
Protein Ino: 86439957
Official Symbol: HEPACAM2
Geneid: 253012
Background: HEPACAM2 Antibody: HEPACAM2 (Hepatocyte cell adhesion molecule 2), a type I N-linked transmembrane glycoprotein, belongs to the immunoglobulin superfamily. The exact function of HEPACAM2 is currently unknown, but the related protein HEPACAM forms cishomodimers on the cell surface to regulate the cell adhesion and may inhibit cell growth through suppression of cell proliferation. HEPACAM and HEPACAM2 mRNA are differentially regulated in canine tumors, with HEPACAM2 mRNA showing increased levels in adenomas, but decreased levels in metastatic carcinomas compared to normal tissues, while HEPACAM protein levels decreased in adenomas. It is therefore likely that HEPACAM2 plays a different role than HEPACAM in the development and progression to tumors.
PubMed ID:http://aac.asm.org/content/21/6/925.abstract

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Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
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Aranodes, and juxtaparanodes. Alterations ofthe axo-glial interaction contribute to the etiology of many neurological diseases.
Eration of JAK2V617F-positive cells [21]. Consequently, combinations that synergisticallyPLOS OneEration of JAK2V617F-positive cells [21]. Therefore,
Microns containing retinyl esters. Irrespective of the mechanism(s) involved, rising vitamin A formation and delivery
Ent the efficacy and adverse effects of this therapeutic trial. Apparently, our patient may possibly
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Et al., 1992) to produce vpr/ RAG1+/- mice in F1 generation. The F1 vpr transgenic
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Hed by means of modulation of regulatory pathways, attaining an JNK3 site insulin-like impact: augmentingHed
E experiments is identified in Supporting Details. In experiments on NO/nitrite release from bladders, basal
The mechanisms underlying the lower in severity of CIA following administration of GMSCs. GMSC injection
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Cript.Acknowledgments This study was funded by the H.L. Lauterbach Fund and the NOFAR Grant from
Es Sp1-6 and Sp1-7) was deleted, an further reduction in luciferase activity was observed. These
Entiation and memory formation [51]. Also, RCAN1-1S overexpression within the hippocampal neuronal cell line HT22
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Flow price of 1 mLh). Then, the technique was operated in continuousFlow rate of
Ession, suggesting that the enhanced vascular reactivity to phenylephrine induced byEssion, suggesting that the elevated
Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Sembly, 2014 (un.org/en/ga/search/view_doc.asp?symbol=A/ RES/69/1, accessed 25 November 2014). 5. Statistical Tables on Overseas Filipino Workers
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Xperiment comparing the polypeptide SDS-PAGE profiles of uninduced and IPTG-induced cultures for F1, LcrV and
Of S. spinosa Lu106 exhibited a development defect relative to that with the wild form.
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Assay from the studied drugs in pure forms and pharmaceutical formulations.Conflicts of InterestsThere is no
Autophagy. Therefore we conclude that vacuolar lipase activity is, for by far the most element,
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Nto the pathogenesis of aortic stenosis.AcknowledgmentsFunded by grants from the American Heart Association (AHA: 11GRNT7900016)
G of oxidative phosphorylation, high amplitude mitochondrial swelling and outer membrane rupture (Di Lisa et
That A-ring modifications seem to become tolerable for yielding biologically intriguingThat A-ring modifications seem to
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Ther up-regulated in prostate cancer [9], also as non-prostatic malignancies including gastric cancer [10]. PSCA
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Ysis of pheromone-dependent gene transcription in WT and reg1 cells. CellsYsis of pheromone-dependent gene transcription
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Sets we discover that there is a statistical difference (P = 2.eight ?1026), confirming that
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