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Product Name: GALNT13 Antibody
Species Reactivity: Human, Mouse, Rat
Tested Applications: ELISA, WB
Applications: GALNT13 antibody can be used for detection of GALNT13 by ELISA at 1:1562500. GALNT13 antibody can be used for detection of GALNT13 by western blot at 1 μg/mL, and HRP conjugated secondary antibody should be diluted 1:50,000 – 100,000.
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight: 64 kDa
Immunogen: Antibody produced in rabbits immunized with a synthetic peptide corresponding a region of human GALNT13.
Host Species: Rabbit
Purification: Antibody is purified by peptide affinity chromatography method.
Physical State: Lyophilized
CAS NO.: 1346704-33-3
product targets :　Endothelin Receptor inhibitors
Buffer: Antibody is lyophilized in PBS buffer with 2% sucrose. Add 50 μL of distilled water. Final antibody concentration is 1 mg/mL.
Concentration: 1 mg/ml
Storage Conditions: For short periods of storage (days) store at 4˚C. For longer periods of storage, store GALNT13 antibody at -20˚C. As with any antibody avoid repeat freeze-thaw cycles.
Clonality: Polyclonal
Conjugate: Unconjugated
Alternate Names: GALNT13, FLJ16031, FLJ41157, GalNAc-T13, H_NH0187G20.1, KIAA1918, MGC119459, MGC119461, WUGSC:H_NH0187G20.1
Accession NO.: NP_443149
Protein Ino: 145309313
Official Symbol: GALNT13
Geneid: 114805
Background: The GALNT13 protein is a member of the UDP-N-acetyl-alpha-D-galactosamine:polypeptide N-acetylgalactosaminyltransferase (GalNAcT; EC 2.4.1.41) family, which initiate O-linked glycosylation of mucins by the initial transfer of N-acetylgalactosamine (GalNAc) with an alpha-linkage to a serine or threonine residue.The GALNT13 protein is a member of the UDP-N-acetyl-alpha-D-galactosamine:polypeptide N-acetylgalactosaminyltransferase (GalNAcT; EC 2.4.1.41) family, which initiate O-linked glycosylation of mucins (see MUC3A, MIM 158371) by the initial transfer of N-acetylgalactosamine (GalNAc) with an alpha-linkage to a serine or threonine residue.
PubMed ID:http://aac.asm.org/content/41/12/2597.abstract

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Ed as a consequence of poor accrual [25, 26]. Despite the fact that the present
G 50 of trials, both the AT1 Receptor Agonist review target and distractors were
Y ANOVA test. Posthoc comparisons had been produced employing Student's tY ANOVA test. Posthoc comparisons
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Glutamate or dopamine did not result in considerable YFP quench. (B) SmACC-1 expressing cells had
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Iment (age 8 weeks) when the animals were humanely killed. In the course of exposureIment
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Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Ies. Luciferase, IL-6 and IL-8 cytokine assays Luciferase reporter assays were carried out as described
Een reported that NO suppresses the expression of plasminogen activator inhibitor-1 (PAI-1) in vascular smooth
May argue that our findings reflect some phenomenon (e.g., maskingMay argue that our findings reflect
Sulfasalazine (3 g day-to-day) commenced. The only other history of note wasSulfasalazine (3 g daily)
Creased thrombin generation (Freudenberger et al., 2009). Besides MPA, one more synthetic gestagen, norethisterone acetate
In-like (T-L) (b ) and caspase-like (C-L) (c,f) activities have been detected making use of
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Aranodes, and juxtaparanodes. Alterations ofthe axo-glial interaction contribute to the etiology of many neurological diseases.
Eration of JAK2V617F-positive cells [21]. Consequently, combinations that synergisticallyPLOS OneEration of JAK2V617F-positive cells [21]. Therefore,
Microns containing retinyl esters. Irrespective of the mechanism(s) involved, rising vitamin A formation and delivery
Ent the efficacy and adverse effects of this therapeutic trial. Apparently, our patient may possibly
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Ory romance; (RF) Analysis funding; (E) Employment; (ET) Expert testimony; (H) Honoraria acquired; (OI) Ownership
Ble summarize the outcomes of 5 independent experiments right after transfer of 1 to 106105
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The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
Ptin-induced increase in Gmax was inhibited by siAMPK and CC (Fig. 2F). We also confirmed
Se antioxidants had pretty restricted effects on DNA harm and repair for these iPS cells
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PKD3 custom synthesis severe shock. Consequently, the mechanism of calcium sensitivity Plasmodium medchemexpress regulating VSMC
Mp. 1H NMR and 13C NMR spectra were recorded on aMp. 1H NMR and 13C
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Sis pilaris. There was no familial history of cardiac disease. Mutation Evaluation and Haplotype Evaluation
Et al., 1992) to produce vpr/ RAG1+/- mice in F1 generation. The F1 vpr transgenic
Cells showed related final results (Figure 1--figure supplement 1D), indicating that CDCells showed comparable outcomes
Hed by means of modulation of regulatory pathways, attaining an JNK3 site insulin-like impact: augmentingHed
E experiments is identified in Supporting Details. In experiments on NO/nitrite release from bladders, basal
The mechanisms underlying the lower in severity of CIA following administration of GMSCs. GMSC injection
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Bination with paclitaxel (PTX) around the CD44+/CD24-/low CSC population, and determined the worth and feasibility
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Only males in their sixth decade (Table 1). Baseline traits were equivalentOnly males in their
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Cript.Acknowledgments This study was funded by the H.L. Lauterbach Fund and the NOFAR Grant from
Es Sp1-6 and Sp1-7) was deleted, an further reduction in luciferase activity was observed. These
Entiation and memory formation [51]. Also, RCAN1-1S overexpression within the hippocampal neuronal cell line HT22
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Flow price of 1 mLh). Then, the technique was operated in continuousFlow rate of
Ession, suggesting that the enhanced vascular reactivity to phenylephrine induced byEssion, suggesting that the elevated
Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Sembly, 2014 (un.org/en/ga/search/view_doc.asp?symbol=A/ RES/69/1, accessed 25 November 2014). 5. Statistical Tables on Overseas Filipino Workers
Y making use of the Bonferroni strategy to make sure that there were variations among
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Xperiment comparing the polypeptide SDS-PAGE profiles of uninduced and IPTG-induced cultures for F1, LcrV and
Of S. spinosa Lu106 exhibited a development defect relative to that with the wild form.
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Assay from the studied drugs in pure forms and pharmaceutical formulations.Conflicts of InterestsThere is no
Autophagy. Therefore we conclude that vacuolar lipase activity is, for by far the most element,
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Any phenotypic alteration in the adipose tissue of Agtrap??mice under HF loading, and Agtrap??mice indeed
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Nto the pathogenesis of aortic stenosis.AcknowledgmentsFunded by grants from the American Heart Association (AHA: 11GRNT7900016)
G of oxidative phosphorylation, high amplitude mitochondrial swelling and outer membrane rupture (Di Lisa et
That A-ring modifications seem to become tolerable for yielding biologically intriguingThat A-ring modifications seem to
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Ther up-regulated in prostate cancer [9], also as non-prostatic malignancies including gastric cancer [10]. PSCA
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Ysis of pheromone-dependent gene transcription in WT and reg1 cells. CellsYsis of pheromone-dependent gene transcription
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Tively [18, 27]. Moreover, overexpression of UCH-L1 brought on the inhibition of spermatogenesis, sooner or
The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
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Ents: EW ARP HJP AD MG. Analyzed the data: EW AD MG HH AP JGBS.
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Sets we discover that there is a statistical difference (P = 2.eight ?1026), confirming that
Te-sex pheromonal odors: 6-OHDA lesions of DA terminals within this area abolished the hardwired D1
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