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Product Name: CLDN13 Antibody
Species Reactivity: Mouse
Tested Applications: ELISA, WB
Applications: CLDN13 antibody can be used for detection of CLDN13 by ELISA at 1:1562500. CLDN13 antibody can be used for detection of CLDN13 by western blot at 1.0 μg/mL, and HRP conjugated secondary antibody should be diluted 1:50,000 – 100,000.
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight: 23 kDa
Immunogen: Antibody produced in rabbits immunized with a synthetic peptide corresponding a region of mouse CLDN13.
Host Species: Rabbit
Purification: Antibody is purified by protein A chromatography method.
Physical State: Lyophilized
CAS NO.: 104931-56-8
Product: Tranilast (Sodium)
Buffer: Antibody is lyophilized in PBS buffer with 2% sucrose. Add 100 μL of distilled water. Final antibody concentration is 1 mg/mL.
Concentration: 1 mg/ml
Storage Conditions: For short periods of storage (days) store at 4˚C. For longer periods of storage, store CLDN13 antibody at -20˚C. As with any antibody avoid repeat freeze-thaw cycles.
Clonality: Polyclonal
Conjugate: Unconjugated
Alternate Names: CLDN13, 2500004B14Rik
Accession NO.: NP_065250
Protein Ino: 10048432
Official Symbol: CLDN13
Geneid: 57255
Background: CLDN13 is a member of claudin family. It is a four-transmembrane protein with WWCC motif, defined by W-X (17-22)-W-X (2)-C-X (8-10)-C.
PubMed ID:http://aac.asm.org/content/36/5/1102.abstract

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Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Ies. Luciferase, IL-6 and IL-8 cytokine assays Luciferase reporter assays were carried out as described
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Creased thrombin generation (Freudenberger et al., 2009). Besides MPA, one more synthetic gestagen, norethisterone acetate
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Aranodes, and juxtaparanodes. Alterations ofthe axo-glial interaction contribute to the etiology of many neurological diseases.
Eration of JAK2V617F-positive cells [21]. Consequently, combinations that synergisticallyPLOS OneEration of JAK2V617F-positive cells [21]. Therefore,
Microns containing retinyl esters. Irrespective of the mechanism(s) involved, rising vitamin A formation and delivery
Ent the efficacy and adverse effects of this therapeutic trial. Apparently, our patient may possibly
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Ory romance; (RF) Analysis funding; (E) Employment; (ET) Expert testimony; (H) Honoraria acquired; (OI) Ownership
Ble summarize the outcomes of 5 independent experiments right after transfer of 1 to 106105
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The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
Ptin-induced increase in Gmax was inhibited by siAMPK and CC (Fig. 2F). We also confirmed
Se antioxidants had pretty restricted effects on DNA harm and repair for these iPS cells
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PKD3 custom synthesis severe shock. Consequently, the mechanism of calcium sensitivity Plasmodium medchemexpress regulating VSMC
Mp. 1H NMR and 13C NMR spectra were recorded on aMp. 1H NMR and 13C
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Et al., 1992) to produce vpr/ RAG1+/- mice in F1 generation. The F1 vpr transgenic
Cells showed related final results (Figure 1--figure supplement 1D), indicating that CDCells showed comparable outcomes
Hed by means of modulation of regulatory pathways, attaining an JNK3 site insulin-like impact: augmentingHed
E experiments is identified in Supporting Details. In experiments on NO/nitrite release from bladders, basal
The mechanisms underlying the lower in severity of CIA following administration of GMSCs. GMSC injection
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T 44 and 38 identity on amino acid level compared with enzymes from
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Bination with paclitaxel (PTX) around the CD44+/CD24-/low CSC population, and determined the worth and feasibility
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Cript.Acknowledgments This study was funded by the H.L. Lauterbach Fund and the NOFAR Grant from
Es Sp1-6 and Sp1-7) was deleted, an further reduction in luciferase activity was observed. These
Entiation and memory formation [51]. Also, RCAN1-1S overexpression within the hippocampal neuronal cell line HT22
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Flow price of 1 mLh). Then, the technique was operated in continuousFlow rate of
Ession, suggesting that the enhanced vascular reactivity to phenylephrine induced byEssion, suggesting that the elevated
Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Sembly, 2014 (un.org/en/ga/search/view_doc.asp?symbol=A/ RES/69/1, accessed 25 November 2014). 5. Statistical Tables on Overseas Filipino Workers
Y making use of the Bonferroni strategy to make sure that there were variations among
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Xperiment comparing the polypeptide SDS-PAGE profiles of uninduced and IPTG-induced cultures for F1, LcrV and
Of S. spinosa Lu106 exhibited a development defect relative to that with the wild form.
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Assay from the studied drugs in pure forms and pharmaceutical formulations.Conflicts of InterestsThere is no
Autophagy. Therefore we conclude that vacuolar lipase activity is, for by far the most element,
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Nto the pathogenesis of aortic stenosis.AcknowledgmentsFunded by grants from the American Heart Association (AHA: 11GRNT7900016)
G of oxidative phosphorylation, high amplitude mitochondrial swelling and outer membrane rupture (Di Lisa et
That A-ring modifications seem to become tolerable for yielding biologically intriguingThat A-ring modifications seem to
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Ethylation in MDA-MB-231 Cells Alterations in DNA methylation by MBD-enriched DNA from MDA-MB-231 cells was
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Ther up-regulated in prostate cancer [9], also as non-prostatic malignancies including gastric cancer [10]. PSCA
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Ysis of pheromone-dependent gene transcription in WT and reg1 cells. CellsYsis of pheromone-dependent gene transcription
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Ng 25 mM exogenous GSH, to identify the existence of passive diffusion of glutathione in
Tively [18, 27]. Moreover, overexpression of UCH-L1 brought on the inhibition of spermatogenesis, sooner or
The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
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Ents: EW ARP HJP AD MG. Analyzed the data: EW AD MG HH AP JGBS.
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Sets we discover that there is a statistical difference (P = 2.eight ?1026), confirming that
Te-sex pheromonal odors: 6-OHDA lesions of DA terminals within this area abolished the hardwired D1
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