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Product Name: CD11c Antibody [N418] (PE-Cy7)
Species Reactivity: Mouse
Tested Applications: Flow
Applications:
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight:
Immunogen:
Host Species: Hamster
Purification: The monoclonal antibody was purified utilizing affinity chromatography and unreacted dye was removed from the product.
Physical State: liquid
CAS NO.: 1151516-14-1
Product: Lesinurad (sodium)
Buffer: Phosphate-buffered aqueous solution, ≤0.09% Sodium azide, may contain carrier protein/stabilizer, ph7.2.
Concentration: 0.2 mg/mL
Storage Conditions: The product should be stored undiluted at 4˚C and should be protected from prolonged exposure to light. Do not freeze.
Clonality: Monoclonal
Conjugate: PE-Cy7
Alternate Names: Cr4, N418, Cd11c, AI449405, Itgax
Accession NO.:
Protein Ino:
Official Symbol: Itgax
Geneid: 16411
Background: The N418 monoclonal antibody specifically reacts with the integrin alphax chain of the mouse CD11c, which is expressed on dendritic cells, CD4-/CD8+ intestinal intraepithelial lymphocytes (IEL) and some activated T lymphocytes. Low levels of CD11c were detected on mouse splenic natural killer cells and on the monocyte/macrophage lineage cells.CD11c expression is upregulated on IEL and T lymphocytes after activation. It binds to CD54, fibrinogen and iC3b and influences the leukocyte adhesive interactions. The N418 antibody binds to CD11c on splenic dendritic cells of the mouse in the T-dependent areas. It also contributes to the binding of iC3b.
PubMed ID:http://aac.asm.org/content/43/12/2881.abstract

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Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Ies. Luciferase, IL-6 and IL-8 cytokine assays Luciferase reporter assays were carried out as described
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Creased thrombin generation (Freudenberger et al., 2009). Besides MPA, one more synthetic gestagen, norethisterone acetate
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Aranodes, and juxtaparanodes. Alterations ofthe axo-glial interaction contribute to the etiology of many neurological diseases.
Eration of JAK2V617F-positive cells [21]. Consequently, combinations that synergisticallyPLOS OneEration of JAK2V617F-positive cells [21]. Therefore,
Microns containing retinyl esters. Irrespective of the mechanism(s) involved, rising vitamin A formation and delivery
Ent the efficacy and adverse effects of this therapeutic trial. Apparently, our patient may possibly
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Ory romance; (RF) Analysis funding; (E) Employment; (ET) Expert testimony; (H) Honoraria acquired; (OI) Ownership
Ble summarize the outcomes of 5 independent experiments right after transfer of 1 to 106105
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The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
Ptin-induced increase in Gmax was inhibited by siAMPK and CC (Fig. 2F). We also confirmed
Se antioxidants had pretty restricted effects on DNA harm and repair for these iPS cells
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PKD3 custom synthesis severe shock. Consequently, the mechanism of calcium sensitivity Plasmodium medchemexpress regulating VSMC
Mp. 1H NMR and 13C NMR spectra were recorded on aMp. 1H NMR and 13C
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Et al., 1992) to produce vpr/ RAG1+/- mice in F1 generation. The F1 vpr transgenic
Cells showed related final results (Figure 1--figure supplement 1D), indicating that CDCells showed comparable outcomes
Hed by means of modulation of regulatory pathways, attaining an JNK3 site insulin-like impact: augmentingHed
E experiments is identified in Supporting Details. In experiments on NO/nitrite release from bladders, basal
The mechanisms underlying the lower in severity of CIA following administration of GMSCs. GMSC injection
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T 44 and 38 identity on amino acid level compared with enzymes from
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Bination with paclitaxel (PTX) around the CD44+/CD24-/low CSC population, and determined the worth and feasibility
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Cript.Acknowledgments This study was funded by the H.L. Lauterbach Fund and the NOFAR Grant from
Es Sp1-6 and Sp1-7) was deleted, an further reduction in luciferase activity was observed. These
Entiation and memory formation [51]. Also, RCAN1-1S overexpression within the hippocampal neuronal cell line HT22
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Flow price of 1 mLh). Then, the technique was operated in continuousFlow rate of
Ession, suggesting that the enhanced vascular reactivity to phenylephrine induced byEssion, suggesting that the elevated
Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Sembly, 2014 (un.org/en/ga/search/view_doc.asp?symbol=A/ RES/69/1, accessed 25 November 2014). 5. Statistical Tables on Overseas Filipino Workers
Y making use of the Bonferroni strategy to make sure that there were variations among
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Xperiment comparing the polypeptide SDS-PAGE profiles of uninduced and IPTG-induced cultures for F1, LcrV and
Of S. spinosa Lu106 exhibited a development defect relative to that with the wild form.
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Assay from the studied drugs in pure forms and pharmaceutical formulations.Conflicts of InterestsThere is no
Autophagy. Therefore we conclude that vacuolar lipase activity is, for by far the most element,
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Nto the pathogenesis of aortic stenosis.AcknowledgmentsFunded by grants from the American Heart Association (AHA: 11GRNT7900016)
G of oxidative phosphorylation, high amplitude mitochondrial swelling and outer membrane rupture (Di Lisa et
That A-ring modifications seem to become tolerable for yielding biologically intriguingThat A-ring modifications seem to
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Ethylation in MDA-MB-231 Cells Alterations in DNA methylation by MBD-enriched DNA from MDA-MB-231 cells was
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Ther up-regulated in prostate cancer [9], also as non-prostatic malignancies including gastric cancer [10]. PSCA
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Ysis of pheromone-dependent gene transcription in WT and reg1 cells. CellsYsis of pheromone-dependent gene transcription
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Ng 25 mM exogenous GSH, to identify the existence of passive diffusion of glutathione in
Tively [18, 27]. Moreover, overexpression of UCH-L1 brought on the inhibition of spermatogenesis, sooner or
The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
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Ents: EW ARP HJP AD MG. Analyzed the data: EW AD MG HH AP JGBS.
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Sets we discover that there is a statistical difference (P = 2.eight ?1026), confirming that
Te-sex pheromonal odors: 6-OHDA lesions of DA terminals within this area abolished the hardwired D1
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