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Product Name: BRS3 Antibody
Species Reactivity: Gibbon, Gorilla, Human
Tested Applications: IHC
Applications: BRS3 antibody can be used in ELISA, Western Blot, immunohistochemistry starting at 10 μg/mL, immunocytochemistry, and immunoprecipitation.
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight:
Immunogen: BRS3 antibody was raised against a peptide located in the 2nd extracellular domain of BRS3 (Human).
Host Species: Rabbit
Purification: Immunoaffinity Chromatography
Physical State: Liquid
CAS NO.: 15176-29-1
Product: Edoxudine
Buffer: PBS, 0.1% sodium azide.
Concentration:
Storage Conditions: BRS3 antibody should be stored long term (months) at -80 ˚C and short term (days) at 4 ˚C. As with all antibodies avoid freeze/thaw cycles.
Clonality: Polyclonal
Conjugate: Unconjugated
Alternate Names: BRS3, BB3 receptor, Bombesin-like receptor 3, Bombesin receptor subtype 3, Bombesin receptor subtype-3, BRS-3, BLP receptor subtype 3, Bombesin receptor 3
Accession NO.: P32247
Protein Ino: 416726
Official Symbol: BRS3
Geneid: 680
Background: BRS3 is classified as an Orphan-A Receptor and is highly similar to the bombesin receptors but has a very low affinity for bombesin itself. Mice lacking BRS3 develop metabolic defects and obesity, suggesting that BRS3 may be an important target for obesity research. In addition, BRS3 may be involved in diabetes and hypertension.
PubMed ID:http://aac.asm.org/content/42/5/1146.abstract

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Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Ies. Luciferase, IL-6 and IL-8 cytokine assays Luciferase reporter assays were carried out as described
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Creased thrombin generation (Freudenberger et al., 2009). Besides MPA, one more synthetic gestagen, norethisterone acetate
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Aranodes, and juxtaparanodes. Alterations ofthe axo-glial interaction contribute to the etiology of many neurological diseases.
Eration of JAK2V617F-positive cells [21]. Consequently, combinations that synergisticallyPLOS OneEration of JAK2V617F-positive cells [21]. Therefore,
Microns containing retinyl esters. Irrespective of the mechanism(s) involved, rising vitamin A formation and delivery
Ent the efficacy and adverse effects of this therapeutic trial. Apparently, our patient may possibly
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Ory romance; (RF) Analysis funding; (E) Employment; (ET) Expert testimony; (H) Honoraria acquired; (OI) Ownership
Ble summarize the outcomes of 5 independent experiments right after transfer of 1 to 106105
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The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
Ptin-induced increase in Gmax was inhibited by siAMPK and CC (Fig. 2F). We also confirmed
Se antioxidants had pretty restricted effects on DNA harm and repair for these iPS cells
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PKD3 custom synthesis severe shock. Consequently, the mechanism of calcium sensitivity Plasmodium medchemexpress regulating VSMC
Mp. 1H NMR and 13C NMR spectra were recorded on aMp. 1H NMR and 13C
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Et al., 1992) to produce vpr/ RAG1+/- mice in F1 generation. The F1 vpr transgenic
Cells showed related final results (Figure 1--figure supplement 1D), indicating that CDCells showed comparable outcomes
Hed by means of modulation of regulatory pathways, attaining an JNK3 site insulin-like impact: augmentingHed
E experiments is identified in Supporting Details. In experiments on NO/nitrite release from bladders, basal
The mechanisms underlying the lower in severity of CIA following administration of GMSCs. GMSC injection
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T 44 and 38 identity on amino acid level compared with enzymes from
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Bination with paclitaxel (PTX) around the CD44+/CD24-/low CSC population, and determined the worth and feasibility
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Cript.Acknowledgments This study was funded by the H.L. Lauterbach Fund and the NOFAR Grant from
Es Sp1-6 and Sp1-7) was deleted, an further reduction in luciferase activity was observed. These
Entiation and memory formation [51]. Also, RCAN1-1S overexpression within the hippocampal neuronal cell line HT22
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Flow price of 1 mLh). Then, the technique was operated in continuousFlow rate of
Ession, suggesting that the enhanced vascular reactivity to phenylephrine induced byEssion, suggesting that the elevated
Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Sembly, 2014 (un.org/en/ga/search/view_doc.asp?symbol=A/ RES/69/1, accessed 25 November 2014). 5. Statistical Tables on Overseas Filipino Workers
Y making use of the Bonferroni strategy to make sure that there were variations among
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Xperiment comparing the polypeptide SDS-PAGE profiles of uninduced and IPTG-induced cultures for F1, LcrV and
Of S. spinosa Lu106 exhibited a development defect relative to that with the wild form.
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Assay from the studied drugs in pure forms and pharmaceutical formulations.Conflicts of InterestsThere is no
Autophagy. Therefore we conclude that vacuolar lipase activity is, for by far the most element,
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Nto the pathogenesis of aortic stenosis.AcknowledgmentsFunded by grants from the American Heart Association (AHA: 11GRNT7900016)
G of oxidative phosphorylation, high amplitude mitochondrial swelling and outer membrane rupture (Di Lisa et
That A-ring modifications seem to become tolerable for yielding biologically intriguingThat A-ring modifications seem to
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Ethylation in MDA-MB-231 Cells Alterations in DNA methylation by MBD-enriched DNA from MDA-MB-231 cells was
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Ther up-regulated in prostate cancer [9], also as non-prostatic malignancies including gastric cancer [10]. PSCA
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Ysis of pheromone-dependent gene transcription in WT and reg1 cells. CellsYsis of pheromone-dependent gene transcription
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Ng 25 mM exogenous GSH, to identify the existence of passive diffusion of glutathione in
Tively [18, 27]. Moreover, overexpression of UCH-L1 brought on the inhibition of spermatogenesis, sooner or
The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
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Ents: EW ARP HJP AD MG. Analyzed the data: EW AD MG HH AP JGBS.
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Sets we discover that there is a statistical difference (P = 2.eight ?1026), confirming that
Te-sex pheromonal odors: 6-OHDA lesions of DA terminals within this area abolished the hardwired D1
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