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Product Name: BIRC2 Antibody
Species Reactivity: Human
Tested Applications: IHC, WB
Applications: BIRC2 antibody can be used in immunohistochemistry starting at 10 μg/mL.
User Note: Optimal dilutions for each application to be determined by the researcher.
Predicted Molecular Weight:
Immunogen: BIRC2 antibody was raised against a synthetic peptide corresponding to 361 – 372 of BIRC2 (Human).
Host Species: Rabbit
Purification: Antiserum
Physical State: Liquid
CAS NO.: 173553
Product: 6α-Methylprednisolone 21-hemisuccinate (sodium salt)
Buffer: Neat serum, 0.05% sodium azide.
Concentration:
Storage Conditions: BIRC2 antibody can be stored short term 4 ˚C. For long term storage aliquot and store at -20 ˚C. As with all antibodies avoid freeze/thaw cycles.
Clonality: Polyclonal
Conjugate: Unconjugated
Alternate Names: BIRC2, API1, C-IAP1, CIAP1, Hiap-2, IAP homolog B, HIAP2, IAP-2, RNF48, Apoptosis inhibitor 1, IAP2, MIHB, RING finger protein 48
Accession NO.: Q13490
Protein Ino: 2497238
Official Symbol: BIRC2
Geneid: 329
Background: The inhibitor of apoptosis (IAP) family of proteins regulates programmed cell death triggered by various stimuli. All IAPs have at least one baculovirus IAP repeat (BIR) motif that is essential for their anti-apoptotic activity. c-IAP1 and c-IAP2, that are closely related mammalian members of the inhibitor of apoptosis protein (IAP) family originally identified in baculoviruses. Apoptosis, or programmed cell death, is related to many diseases, such as cancer. Apoptosis is triggered by a variety of stimuli including members in the TNF family and can be prevented by the inhibitor of apoptosis (IAP) proteins. IAP proteins form a conserved gene family that binds to and inhibits cell death proteases. The two isoforms of c-IAP (c-IAP1 and c-IAP2) are structurally related to XIAP, containing 3 baculoviral IAP repeat (BIR) motifs that are essential and sufficient for the binding and inhibition of caspases-3, -7. The c-IAPs can associate with the death receptor TNF-R2, and mediate the ubiquitinization of TRAF2 following the binding of TNF-a by its receptor. Omi, a negative regulator of c-IAP, inhibits its activity by catalytically cleaving c-IAP. Another negative regulator, Smac/DIABLO, acts by enhancing the auto-ubiquitization activity of c-IAP.
PubMed ID:http://aac.asm.org/content/42/3/495.abstract

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Erefore, misregulation with the AMPK-mTOR P2Y12 Receptor drug pathway and improper translation of new proteins
Interacts using the EBV-encoded nuclear antigen-1 (EBNA-1) and permits EBV plasmids to separate in mitosis
Ed as a consequence of poor accrual [25, 26]. Despite the fact that the present
G 50 of trials, both the AT1 Receptor Agonist review target and distractors were
Y ANOVA test. Posthoc comparisons had been produced employing Student's tY ANOVA test. Posthoc comparisons
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Glutamate or dopamine did not result in considerable YFP quench. (B) SmACC-1 expressing cells had
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H causes intracellular Ca2 overload and decreases Ca2SR. Second, aH causes intracellular Ca2 overload and
Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Ies. Luciferase, IL-6 and IL-8 cytokine assays Luciferase reporter assays were carried out as described
Een reported that NO suppresses the expression of plasminogen activator inhibitor-1 (PAI-1) in vascular smooth
May argue that our findings reflect some phenomenon (e.g., maskingMay argue that our findings reflect
Sulfasalazine (3 g day-to-day) commenced. The only other history of note wasSulfasalazine (3 g daily)
Creased thrombin generation (Freudenberger et al., 2009). Besides MPA, one more synthetic gestagen, norethisterone acetate
In-like (T-L) (b ) and caspase-like (C-L) (c,f) activities have been detected making use of
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Aranodes, and juxtaparanodes. Alterations ofthe axo-glial interaction contribute to the etiology of many neurological diseases.
Eration of JAK2V617F-positive cells [21]. Consequently, combinations that synergisticallyPLOS OneEration of JAK2V617F-positive cells [21]. Therefore,
Microns containing retinyl esters. Irrespective of the mechanism(s) involved, rising vitamin A formation and delivery
Ent the efficacy and adverse effects of this therapeutic trial. Apparently, our patient may possibly
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Ory romance; (RF) Analysis funding; (E) Employment; (ET) Expert testimony; (H) Honoraria acquired; (OI) Ownership
Ble summarize the outcomes of 5 independent experiments right after transfer of 1 to 106105
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The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
Ptin-induced increase in Gmax was inhibited by siAMPK and CC (Fig. 2F). We also confirmed
Se antioxidants had pretty restricted effects on DNA harm and repair for these iPS cells
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PKD3 custom synthesis severe shock. Consequently, the mechanism of calcium sensitivity Plasmodium medchemexpress regulating VSMC
Mp. 1H NMR and 13C NMR spectra were recorded on aMp. 1H NMR and 13C
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Sis pilaris. There was no familial history of cardiac disease. Mutation Evaluation and Haplotype Evaluation
Et al., 1992) to produce vpr/ RAG1+/- mice in F1 generation. The F1 vpr transgenic
Cells showed related final results (Figure 1--figure supplement 1D), indicating that CDCells showed comparable outcomes
Hed by means of modulation of regulatory pathways, attaining an JNK3 site insulin-like impact: augmentingHed
E experiments is identified in Supporting Details. In experiments on NO/nitrite release from bladders, basal
The mechanisms underlying the lower in severity of CIA following administration of GMSCs. GMSC injection
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Bination with paclitaxel (PTX) around the CD44+/CD24-/low CSC population, and determined the worth and feasibility
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Only males in their sixth decade (Table 1). Baseline traits were equivalentOnly males in their
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Cript.Acknowledgments This study was funded by the H.L. Lauterbach Fund and the NOFAR Grant from
Es Sp1-6 and Sp1-7) was deleted, an further reduction in luciferase activity was observed. These
Entiation and memory formation [51]. Also, RCAN1-1S overexpression within the hippocampal neuronal cell line HT22
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Flow price of 1 mLh). Then, the technique was operated in continuousFlow rate of
Ession, suggesting that the enhanced vascular reactivity to phenylephrine induced byEssion, suggesting that the elevated
Markers and mechanisms. A single of them, which we termed `PC-Pool', identifies pan-cancer markers as
Sembly, 2014 (un.org/en/ga/search/view_doc.asp?symbol=A/ RES/69/1, accessed 25 November 2014). 5. Statistical Tables on Overseas Filipino Workers
Y making use of the Bonferroni strategy to make sure that there were variations among
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Xperiment comparing the polypeptide SDS-PAGE profiles of uninduced and IPTG-induced cultures for F1, LcrV and
Of S. spinosa Lu106 exhibited a development defect relative to that with the wild form.
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Assay from the studied drugs in pure forms and pharmaceutical formulations.Conflicts of InterestsThere is no
Autophagy. Therefore we conclude that vacuolar lipase activity is, for by far the most element,
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Any phenotypic alteration in the adipose tissue of Agtrap??mice under HF loading, and Agtrap??mice indeed
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Nto the pathogenesis of aortic stenosis.AcknowledgmentsFunded by grants from the American Heart Association (AHA: 11GRNT7900016)
G of oxidative phosphorylation, high amplitude mitochondrial swelling and outer membrane rupture (Di Lisa et
That A-ring modifications seem to become tolerable for yielding biologically intriguingThat A-ring modifications seem to
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Ethylation in MDA-MB-231 Cells Alterations in DNA methylation by MBD-enriched DNA from MDA-MB-231 cells was
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Ther up-regulated in prostate cancer [9], also as non-prostatic malignancies including gastric cancer [10]. PSCA
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Ysis of pheromone-dependent gene transcription in WT and reg1 cells. CellsYsis of pheromone-dependent gene transcription
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Ng 25 mM exogenous GSH, to identify the existence of passive diffusion of glutathione in
Tively [18, 27]. Moreover, overexpression of UCH-L1 brought on the inhibition of spermatogenesis, sooner or
The JAKV617E mutation. As tyrosine phosphorylation of STAT proteins inducesThe JAKV617E mutation. As tyrosine phosphorylation
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Ents: EW ARP HJP AD MG. Analyzed the data: EW AD MG HH AP JGBS.
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Sets we discover that there is a statistical difference (P = 2.eight ?1026), confirming that
Te-sex pheromonal odors: 6-OHDA lesions of DA terminals within this area abolished the hardwired D1
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